Algodystrophy: Diagnosis and Therapy of a Frequent Disease by Professeur Paul Doury, Dr. Yves Dirheimer, Dr. Serge Pattin

By Professeur Paul Doury, Dr. Yves Dirheimer, Dr. Serge Pattin (auth.)

It is with nice excitement and ,much curiosity that I approved to write down the foreword to this booklet by way of Paul Doury, Yves Dirheimer, and Serge Pattin almost about "algodystrophy." First, simply because i do know the level in their own adventure, from which they've got chosen the simplest for this e-book. moment, since it appeared to me that their targeted research of the varied works at the topic, works that have been released worldwide and which offer various physiopathologic interpretations, would supply a complete research assembly a true want. Algodystrophy, to undertake the time period utilized by the authors, advantages rheumatolog­ ists' cautious cognizance. it's certainly a common and, as is now renowned, happens within the such a lot different etiologic situations; it's not exclusively posttraumatic, a idea on which analysis had lengthy been established. This variable etiology indicates the complexity of algodystrophy's pathogenic mechanism.

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Extra resources for Algodystrophy: Diagnosis and Therapy of a Frequent Disease of the Locomotor Apparatus

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Irregular spotty hypertranslucency of the bone shadows, producing a striking peculiar dappled image. " Later these features were seen by all workers; they consist usually of individualized rounded or oval images without precise limits, present in large amounts. Not uncommonly there are also more diffuse demineralization areas, giving the impression that the bone architecture has been irregularly rubbed out, or on the contrary there may be smaller, punctiform images (Fig. 13). The anomalies of the bone architecture sometimes have other appearances, which may exist alone or be associated to produce the background for vacuolar osteoporosis.

We summarize below the results of our first 24 studies bearing on locus A and B. 5%; BW15, 12%. There is no marked difference between these results and our own short series. No conclusion can however be drawn from such fragmentary results, which need to be confirmed by subsequent studies. G. , containing less than 500 cells/mm 3 . In our series of 250 cases the synovial fluid was examined in 12 cases and shown to contain few cells. H. Muscular Enzymes To our knowledge no studies have reported changes in the muscular enzymes in algodystrophy.

2. Clinical Signs Pain is the cardinal symptom, but its mode of occurrence, site, and character vary greatly. The onset is often sudden (in 6 of Lequesne's 10 cases [473] and in 6 of Hunder's 9 cases [382]). The onset is sometimes so well-defined that the patient is able to indicate the precise moment of its occurrence and thus to correlate it to a slight injury or to muscle strain. According to Grosclaude [331] pain more often develops progressively. Whatever the mode of onset, the pain becomes worse and reaches maximum intensity after several days to 1 month, sometimes 2-3 months.

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